カテゴリ分類されていないページ

移動: 案内検索

1 件目から100 件目までの範囲内で最大 100 件の結果を表示しています。

(前の100件 | 次の100件) (20 | 50 | 100 | 250 | 500 件) を表示

  1. "Golden Suggestions": Some Tips For Rare metal Promoting, Acquiring, And A Lot More
  2. "Tillers,"in Science with the Rice Plant: Quantity A person Morphology, eds
  3. " has any bearing on her illness or the way she copes.
  4. &
  5. 's protocol. Full RNA was calculated, and 1 to 5 g were being reverse
  6. 's protocol. Total RNA was measured, and one to 5 g had been reverse
  7. (0.05) and 96 h (0.14) (Fig. 1i, j). Assessment of time course by means of Student
  8. (10), exactly where an progressed AGT variant is precisely labeled in vivo by
  9. (10), in which an advanced AGT variant is specially labeled in vivo by
  10. (10), where an evolved AGT variant is particularly labeled in vivo by
  11. (10), where an progressed AGT variant is specifically labeled in vivo by
  12. (10), where by an developed AGT variant is exclusively labeled in vivo by
  13. (10), wherever an advanced AGT variant is precisely labeled in vivo by
  14. (10), wherever an evolved AGT variant is specifically labeled in vivo by
  15. (10, 56). Hence, to determine much more specially the extent of sequence similarity among
  16. (10, fifty six). Hence, to ascertain a lot more exclusively the extent of sequence similarity between
  17. (10, fifty six). So, to find out additional specifically the extent of sequence similarity in between
  18. (18). Both of these techniques, coinfection with adenovirus and knockdown of U2 snRNP
  19. (18). These two methods, coinfection with adenovirus and knockdown of U2 snRNP
  20. (18). These two ways, coinfection with adenovirus and knockdown of U2 snRNP
  21. (2006) mutations map for the inside of in the channel, ruling out their
  22. (2006) mutations map for the inside of of your channel, ruling out their
  23. (2006) mutations map for the within on the channel, ruling out their
  24. (2006) mutations map into the inside of in the channel, ruling out their
  25. (2006) mutations map into the inside with the channel, ruling out their
  26. (2006) mutations map on the inside of in the channel, ruling out their
  27. (2006) mutations map on the within on the channel, ruling out their
  28. (2006) mutations map to the inside of of your channel, ruling out their
  29. (7) days and have been euthanized by CO2 asphyxiation on indications of systemic
  30. (7) demonstrates that membrane asymmetry is an vital mediator in the regulatory
  31. (7) demonstrates that membrane asymmetry is definitely an crucial mediator with the regulatory
  32. (7) demonstrates that membrane asymmetry is definitely an essential mediator from the regulatory
  33. (7) demonstrates that membrane asymmetry is definitely an essential mediator of your regulatory
  34. (7) times and have been euthanized by CO2 asphyxiation upon signs of systemic
  35. (7) times and have been euthanized by CO2 asphyxiation upon symptoms of systemic
  36. (Cubozoa, Carybdeidae) in die Meduse. Helgol Meeresunters 1985, 39:129-164. 115. Land MF: Animal
  37. (Cubozoa, Carybdeidae) in die Meduse. Helgol Meeresunters 1985, 39:129-164. a hundred and fifteen. Land MF: Animal
  38. (Cubozoa, Carybdeidae) in die Meduse. Helgol Meeresunters 1985, 39:129-164. one hundred fifteen. Land MF: Animal
  39. (Fig. 3f, Supplementary Fig. S4). Below these problems, PatA, also to
  40. (Fig. 3f, Supplementary Fig. S4). Less than these situations, PatA, and to
  41. (Fig. 4c). To investigate the part of resveratrol on AMPK, cells
  42. (Figure 3a). When proteins are degraded at the post-translational level, there
  43. (GAPs) these types of as 2- and 2-chimaerin, EphA receptor activation sales opportunities to
  44. (KSOM; EMD Millipore, Billerica, MA) supplemented with 0.3 fatty acid free bovine
  45. (Lewen et al., 2000). Our conclusions indicate that Rit-dependent signaling is actually a
  46. (Meiklejohn et al. 2013). Collectively, these mutations disrupt larval metabolism, delay improvement
  47. (Meiklejohn et al. 2013). Together, these mutations disrupt larval metabolism, delay improvement
  48. (NMDG-Glu) answer employed as an external remedy contained (in mM): 120 NMDG
  49. (PDH) metabolism and had been probable cofractionated since in their association with
  50. (Perkin-Elmer LS50B; PerkinElmer Life and Analytical Sciences, Waltham, MA, USA
  51. (Vasseur et al., 2002a). p8 Represses FoxO3 Transactivation of bnip3 p
  52. (Warburton et al., 1986).M.W. Brown et al. / Neuropsychologia 50 (2012) 31224. Item recognition
  53. (autophagosomes), in parallel having a substantial reduce in pink puncta, confirming
  54. (eighteen). These two strategies, coinfection with adenovirus and knockdown of U2 snRNP
  55. (ii) robust neurodegeneration is present, and (iii) animals haven't nonetheless
  56. (jewelry/jewellery )
  57. (left) and inside the presence of 200 M AITC (right). (B) Summary
  58. (mutant peak is indicated by a grey arrow). The bases are
  59. (n = 15), srt mutant (n = twenty) and srt complemented strains (n = ten). Disease progression
  60. (n = fifteen), srt mutant (n = 20) and srt complemented strains (n = ten). Ailment development
  61. (n = fifteen), srt mutant (n = 20) and srt complemented strains (n = ten). Condition progression
  62. (n = fifteen), srt mutant (n = 20) and srt complemented strains (n = ten). Disease development
  63. (n = fifteen), srt mutant (n = 20) and srt complemented strains (n = ten). Disease progression
  64. (n = fifteen), srt mutant (n = 20) and srt complemented strains (n = ten). Illness progression
  65. (n = fifteen), srt mutant (n = twenty) and srt complemented strains (n = 10). Disorder development
  66. (n = fifteen), srt mutant (n = twenty) and srt complemented strains (n = 10). Sickness development
  67. (n = fifteen), srt mutant (n = twenty) and srt complemented strains (n = ten). Ailment progression
  68. (n = fifteen), srt mutant (n = twenty) and srt complemented strains (n = ten). Disease progression
  69. (pH 5.five, containing 0.five mM EDTA, 2 mM TCEP, and 0.two LPPG). Near-UV and Far-UV
  70. (pH 7.5) buffer that contains 0.twenty five M sucrose, one mM EDTA, 10 mM -mercaptoethanol, 1 mM benzamidine
  71. (rings/engagement rings)
  72. (seven) days and ended up euthanized by CO2 asphyxiation on signs of systemic
  73. (seven) days and ended up euthanized by CO2 asphyxiation upon indicators of systemic
  74. (seven) days and had been euthanized by CO2 asphyxiation upon signals of systemic
  75. (seven) times and ended up euthanized by CO2 asphyxiation on indications of systemic
  76. (seven) times and have been euthanized by CO2 asphyxiation on symptoms of systemic
  77. (seven) times and were being euthanized by CO2 asphyxiation on signs of systemic
  78. (ten), exactly where an developed AGT variant is exclusively labeled in vivo by
  79. (ten), where an evolved AGT variant is especially labeled in vivo by
  80. (ten), where an evolved AGT variant is particularly labeled in vivo by
  81. (ten), where by an advanced AGT variant is specially labeled in vivo by
  82. (ten), wherever an progressed AGT variant is precisely labeled in vivo by
  83. (ten, 56). As a result, to determine a lot more precisely the extent of sequence similarity amongst
  84. (ten, fifty six). Consequently, to determine much more particularly the extent of sequence similarity in between
  85. (ten, fifty six). Hence, to determine a lot more specifically the extent of sequence similarity in between
  86. (ten, fifty six). Hence, to determine far more especially the extent of sequence similarity between
  87. (ten, fifty six). Thus, to ascertain far more precisely the extent of sequence similarity in between
  88. ), but their role in presynaptic assembly continues to be unclear. Also, the detection
  89. ), namely HPCAL1, HPCAL4, NCALD, ARRB1, PDE6D, and STK16. We in comparison
  90. ), namely HPCAL1, HPCAL4, NCALD, ARRB1, PDE6D, and STK16. We in contrast
  91. ), namely HPCAL1, HPCAL4, NCALD, ARRB1, PDE6D, and STK16. We when compared
  92. ), particularly HPCAL1, HPCAL4, NCALD, ARRB1, PDE6D, and STK16. We compared
  93. ), particularly HPCAL1, HPCAL4, NCALD, ARRB1, PDE6D, and STK16. We in contrast
  94. ), specifically HPCAL1, HPCAL4, NCALD, ARRB1, PDE6D, and STK16. We compared
  95. ), which can encourage renal fibrosis by means of produced lipid peroxides (Neau et
  96. ), which can encourage renal fibrosis by way of created lipid peroxides (Neau et
  97. ), which can promote renal fibrosis by way of created lipid peroxides (Neau et
  98. ), which could encourage renal fibrosis through generated lipid peroxides (Neau et
  99. ), which could stimulate renal fibrosis by using created lipid peroxides (Neau et
  100. ), which may promote renal fibrosis by using created lipid peroxides (Neau et

(前の100件 | 次の100件) (20 | 50 | 100 | 250 | 500 件) を表示